Do you know if you are suffering from Chronic Candida? It can be very easy to tell. That is why we have developed this important questionnaire.

This questionnaire is designed for adults and the scoring system isn't appropriate for children. It lists factors in your medical history which promote the growth of Candida Albicans (Section A), and symptoms commonly found in individuals with yeast-connected illness (Sections B and C).

What is Candida?

Candida problems are thought to be caused when the beneficial yeast form of candida albicans mutates to it's fungal form. Candida can take over sections of our intestinal tract causing numerous symptoms. As it grows out of balance it is said to produce toxins which affect the mucousal lining of the intestines leading to leaky gut syndrome and a whole host of problems. Many aggravating daily symptoms may occur as well as auto-immune diseases and Inflammed Bowel Diseases. Candida Albicans is a yeast infection, both digestive and sometimes systemic. Literally millions of men and women have a potential yeast infection that are causing a significant number of health problems or conditions. It has become the missing link in disease as well as the most under-diagnosed (ignored) problem in modern medicine.

It is estimated to affect 30 million men and women everyday. It is also estimated that nearly half the world's population has or will have a moderate or severe Candida problem at some point in their lives.

Candida albicans (fungi) normally resides in the intestinal tract along with (friendly) bacteria such as, E. Coli, Lactobacillus Acidophilus, Lactobacillus Bifidus, Enterococci, Coliforms, etc. In proper balance they aid in the digestion of food and the production of vitamins without causing harm. The use of antibiotics and other bacteriostatic agents can destroy the friendly bacterial flora of the intestinal tract without harming the fungi (Candida albicans). Without the presence of significant bacterial flora, Candida albicans can grow without control.

Once overgrowth of Candida albicans has occurred in the intestinal tract it forms myceles, which are spike-like structures that allow it to penetrate the intestinal walls and enter the bloodstream. The intestinal walls become damaged from the myceles leaving holes in the walls. After entering the blood, Candida albicans causes an inflammatory immune system response. Additionally, whole proteins and other large molecules can pass through the holes in the damaged intestinal walls, referred to as Increased Intestinal Permeability or Leaky Gut Syndrome, causing further allergic reactions to occur. It may take many years to repair the damage to the intestinal walls.

In the bloodstream, Candida albicans can mutate to a cell wall deficient form and (hide) from a weak immune system. In the cell wall deficient form there are no cell wall antigens to provoke an immune reaction with fever. It is possible to have cell wall deficient microorganisms for a lifetime if not propeily treated.

Recent research is indicating a complication of intestinal Candida albicans overgrowth, as a result of antibiotic therapy, with an associated intestinal overgrowth of the bacteria Clostridium difficile. There are also many bacteria that enter the stomach from the food supply. If an individual is experiencing stress, that individual may not produce enough hydrochloric acid to destroy microorganisms that enter the stomach from the food supply. 1-lelicobacter pylori is a typical bacteria that invades the stomach and may eventually cause an ulceration in the stomach or intestinal lining. Other food-born (non-friendly) bacteria that may enter the digestive track are salmonella, shigella, and yersinia, which can also become cell wall deficient.

Normally, the cell wall membranes of bacteria and fungi contain antigen codes, which are detected by the immune system. The immune system in turn produces antibodies to match the antigens of the bacteria or fungi. The antibodies affact to the microorganisms and destroy them. These new forms of microorganisms are lacking complete cell wall membranes. The immune system will not produce antibodies because it didn't detect an antigen code. Nor will the immune system raise the body temperature, which is normally the first sign of infection. Normal antibody blood tests will not detect the presence of such microorganisms because they didn't produce antibodies. Antibiotics are ineffective (antibiotic resistance) because the antibiotics can't attach to a cell wall antigen.

The 7 stages of the 'inflamed' gut.

1 . When the gut is inflamed, it does not absorb nutrients and foods properly and so fatigue and bloating can occur.

2. As mentioned previously, when large food particles are absorbed there is the creation of food allergies and new symptoms.

3. When the gut is inflamed the carrier proteins are damaged so nutrient deficiencies can occur.

4. Likewise when the detoxification pathways that line the gut are compromised, chemical sensitivity can arise. Furthermore the leakage of toxins overburdens the liver so that the body is less able to handle everyday chemicals.

5. When the gut lining is inflamed the protective coating of lgA (immunoglobulin A) is adversely affected and the body is not able to ward off protozoa, bacteria, viruses and yeasts.

6. When the intestinal lining is inflamed, bacteria and yeasts are able to trans-locate. This means that they are able to pass from the gut lumen or cavity, into the bloodstream and set up infection anywhere else in the body.

7. The worst symptom is the formation of antibodies. Sometimes these leak across and look similar to antigens on our own tissues. Consequently, when an antibody is made to attack it, it also attacks our tissue. This is probably how autoimmune disease start.

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